Rip
glad i never started it
glad i never started it
a story isnt just about the conclusion, its about the journey
berserk is an experiece breh
Def the most influential manga of all time. Everything in the ‘Dark Fantasy’ genre borrows from it
Non-artist wont understand how crazy this is for a single pan in a manga
Acute aortic dissectionHe died 2 weeks ago and the family is telling us now
![]()
Aortic dissection is the separation of the aorta into two areas of blood flow, - the true and false lumen held apart by an arterial flap resulting from the tear. In the false lumen, interrupted blood flow can compromise blood flow in the true lumen due to compression, and when the two lumens communicate due to re-entry, blood flow is present in both but can cause aneurysmal degeneration. Rupture, extensive hemorrhage, aortic valve incompetence, coronary dissection, cardiac tamponade, stroke and malperfusion are likely complications of acute aortic dissection. Surgery will usually involve placing a Dacron graft; medication will be a combination of nitrates and beta-blockers.
Symptoms can vary according to where the tear is localised, and where it migrates to. Tear in the ascending aorta would bring on pain in the anterior, midline chest. A tear in the aortic arch might bring on pain in the jaw or neck. Intra scapular back and abdominal pain are more common with the descending aorta.
Intramural hematoma, symptomatic aortic ulcer, and symptomatic aneurysm syndrome together with acute aortic dissection traditionally make up the wider spectrum of aortic disease. However, intramural hematoma and hemorrhage, and aortic ulcers can be seen as signs of evolving dissections according to the 2001 ESC guidelines on aortic dissection, which offered 5 classes of aortic dissection according to that staging of the disease: 1) classical (intimal flap between true and false lumen), 2) medial disruption (with formation of intramural haemotoma), 3) subtle dissection (without haematoma, eccentric bulge at tear site) 4) rupture (leading to aortic ulceration, penetrating aortic atherosclerotic ulcer with surrounding haemoatoma usually sub adventitial), or 5) iatrogenic and traumatic.
With onset of symptoms as reference point, aortic dissection is acute within two weeks; sub acute from 2 to 6 weeks and it is chronic beyond 6 weeks.
Previous to the ESC's disease staging, two anatomic systems widely served to describe it: 1) The Stanford system involving the ascending artery (type A) or not (type B) and 2) the DeBakey system which refers to the original site of intimal tear: I - ascending aorta propagating to aortic arch and II - ascending arch only and III - descending aorta and extending distally or proximally
A third, descriptive classification refers to proximal aortic dissection: (Stanford type A or DeBakey Type I and II 2) or distal: Stanford Type B or DeBakey Type III. (Fig. 1)
Cardiovascular morbidity and mortality associated with this condition are high. Hospital-based mortality rates are approximately 30%. Patients with type A aortic dissection who undergo surgical treatment have a 30% mortality rate; patients who receive medical treatment have a mortality rate of 60%. The annual incidence of aortic dissection - 5 to 30 per million is low compared to that of myocardial infarction which is 4,000 per million.